GCM2

Glial cells missing homolog 2 (Drosophila), also known as GCM2, is a human gene.cite web | title = Entrez Gene: GCM2 glial cells missing homolog 2 (Drosophila)| url = http://www.ncbi.nlm.nih.gov/sites/entrez?Db=gene&Cmd=ShowDetailView&TermToSearch=9247| accessdate = ]

PBB_Summary
section_title =
summary_text = The Drosophila 'glial cells missing' (gcm) gene is thought to act as a binary switch between neuronal and glial cell determination. The gcm protein and mammalian gcm homologs contain a conserved N-terminal gcm motif that has DNA-binding activity. See GCM1 (MIM 603715). [supplied by OMIM] cite web | title = Entrez Gene: GCM2 glial cells missing homolog 2 (Drosophila)| url = http://www.ncbi.nlm.nih.gov/sites/entrez?Db=gene&Cmd=ShowDetailView&TermToSearch=9247| accessdate = ]

References

Further reading

PBB_Further_reading
citations =
*cite journal | author=Kanemura Y, Hiraga S, Arita N, "et al." |title=Isolation and expression analysis of a novel human homologue of the Drosophila glial cells missing (gcm) gene. |journal=FEBS Lett. |volume=442 |issue= 2-3 |pages= 151–6 |year= 1999 |pmid= 9928992 |doi=
*cite journal | author=Kammerer M, Pirola B, Giglio S, Giangrande A |title=GCMB, a second human homolog of the fly glide/gcm gene. |journal=Cytogenet. Cell Genet. |volume=84 |issue= 1-2 |pages= 43–7 |year= 1999 |pmid= 10343099 |doi=
*cite journal | author=Correa P, Akerström G, Westin G |title=Underexpression of Gcm2, a master regulatory gene of parathyroid gland development, in adenomas of primary hyperparathyroidism. |journal=Clin. Endocrinol. (Oxf) |volume=57 |issue= 4 |pages= 501–5 |year= 2002 |pmid= 12354132 |doi=
*cite journal | author=Strausberg RL, Feingold EA, Grouse LH, "et al." |title=Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences. |journal=Proc. Natl. Acad. Sci. U.S.A. |volume=99 |issue= 26 |pages= 16899–903 |year= 2003 |pmid= 12477932 |doi= 10.1073/pnas.242603899
*cite journal | author=Mungall AJ, Palmer SA, Sims SK, "et al." |title=The DNA sequence and analysis of human chromosome 6. |journal=Nature |volume=425 |issue= 6960 |pages= 805–11 |year= 2003 |pmid= 14574404 |doi= 10.1038/nature02055
*cite journal | author=Maret A, Bourdeau I, Ding C, "et al." |title=Expression of GCMB by intrathymic parathyroid hormone-secreting adenomas indicates their parathyroid cell origin. |journal=J. Clin. Endocrinol. Metab. |volume=89 |issue= 1 |pages= 8–12 |year= 2004 |pmid= 14715818 |doi=
*cite journal | author=Gerhard DS, Wagner L, Feingold EA, "et al." |title=The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC). |journal=Genome Res. |volume=14 |issue= 10B |pages= 2121–7 |year= 2004 |pmid= 15489334 |doi= 10.1101/gr.2596504
*cite journal | author=Kebebew E, Peng M, Wong MG, "et al." |title=GCMB gene, a master regulator of parathyroid gland development, expression, and regulation in hyperparathyroidism. |journal=Surgery |volume=136 |issue= 6 |pages= 1261–6 |year= 2005 |pmid= 15657585 |doi= 10.1016/j.surg.2004.06.056
*cite journal | author=Thomée C, Schubert SW, Parma J, "et al." |title=GCMB mutation in familial isolated hypoparathyroidism with residual secretion of parathyroid hormone. |journal=J. Clin. Endocrinol. Metab. |volume=90 |issue= 5 |pages= 2487–92 |year= 2005 |pmid= 15728199 |doi= 10.1210/jc.2004-2450
*cite journal | author=Baumber L, Tufarelli C, Patel S, "et al." |title=Identification of a novel mutation disrupting the DNA binding activity of GCM2 in autosomal recessive familial isolated hypoparathyroidism. |journal=J. Med. Genet. |volume=42 |issue= 5 |pages= 443–8 |year= 2006 |pmid= 15863676 |doi= 10.1136/jmg.2004.026898

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